Lyme Cover-up

Regarding the Lyme surveillance system, you always have at least a tenfold under-reporting. And the CDC even admits Lyme is an epidemic. At least 300,000 people are infected with Lyme disease each year in the United States and that number is grossly underestimated; 10 times than what was first reported! That is 25,000 new cases per month. It is more prevalent than HIV, breast cancer and AIDS. The CDC in their own words states that it is the FASTEST GROWING INFECTIOUS DISEASE in the US.

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Multinationals decide which researchers get the cash. Researchers wanting to test the postulation of bacterial etiology of MS are shunned as if they were crackpots. Big Pharma makes billions a year on MS symptom relievers and they trickle millions down to their footsoldiers, the “MS experts”. A cure would be a severe financial blow. Even more so, because there is strong evidence that many other neurological illnesses are caused by germs as well. Because due to the phenomenon of immune privilege there is an inadequate immune response in the brain and spinal cord, making these organs the ideal place for certain slow-dividing spirochetal bacteria to entrench, multiply and cause lesions. The entire concept of antibiotic-resistant, hard-to-test-for chronic CNS infections leading to a plethora of neurological syndromes has to be suppressed and what can’t be suppressed will be craftily discredited. Better to give every expression of a neurological infection its own name such as “MS”, “Alzheimers”, “Parkinsons,” “ALS” and “Fibromyalgia”. And fund armies of ignorant “experts” to obfuscate the issue, whilst boycotting, firing, censoring, smearing and suing those few real experts that refuse to stay in line. Big Pharma is in business to make money, and money is made when people are ill, not when they’re healthy. Anyone standing in their way is relegated to the sidelines. Patents are being bought and shelved so that cures will never see the light of day.

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Even Publications trying to debunk the spirochetal etiology of MS had to face the inconvenient facts; Read here: 

http://www.owndoc.com/uploads/ms-spirochetes.png

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1986 (USA): Relapsing fever/Lyme disease – Multiple sclerosis. Medical Hypotheses, volume 21, issue 3,  pages 335-343 

Synopsis: In MS, the plaques have their origin around veins in the central nervous system. This corresponds with the lesions found in neuroborreliosis. The geographical spread of MS correlates strongly with mean annual temperature. The geographical distribution of the ticks that transmit Lyme disease have a similar geographical distribution. There have been MS “epidemics” in the past where 40 times more cases of MS occurred than normal. Those epidemics appear to correlate with the large-scale introduction of dogs or other animals that are hosts for ticks. Postulates that Borrelia spirochetes may be acting as the trigger in MS, setting off an autoimmune reaction in which patients produce antibodies that attack their own nerve fibers. Mentions that Borrelia eat myelin as well. Notes how many particular and peculiar Lyme symptoms are shared with MS symptoms. Mentions a small study in which two out of eight MS patients tested positive for Lyme disease.

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2000 (Poland): Lyme borreliosis and Multiple sclerosis: Any Connection? A Seroepidemic study. Ann Agric Environ Med. issue 7, 141-143

Synopsis: 10 out of 26 MS patients tested positive for Lyme borreliosis. Notes how it is virtually impossible to make a distinction between late stage Lyme disease and Multiple sclerosis, not even with MRI. Diagnosis of MS vs. late stage neuroborreliosis are guesswork – there are no reliable tests for either. Conclusion: Multiple sclerosis may often be associated with Borrelia infection.

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2001 (Norway): Association between Multiple sclerosis and Cystic Structures in Cerebrospinal Fluid. Infect 29:315 

Synopsis: Borrelia cysts were found in all ten out of ten patients diagnosed with Multiple Sclerosis. No bacteria were found in a control group. The most modern methods such as a transmission electron microscope were used by a specialist in this narrow field – this may explain why a 100% infection rate was found by Brorson, as opposed to lower rates in other research. The cysts turned into spirochetal bacteria when cultured. Remarks that the bacterial infection theory of MS was abandoned because antibiotics did not help. Remarks that Borrelia bacteria have mechanisms to evade the immune system and survive antibiotics, and offers research evidence for that. Concludes that all ten MS patients have been infected with a spirochete. Dismisses the common criticism that “all those MS patients were also infected with an unrelated Lyme disease” by pointing out how unlikely that is, especially seen the ample research evidence for a spirochetal cause of MS. Concludes that MS could very well be a chronic infection. Points out that there is microbiological and clinical evidence that spirochetal bacteria could be the cause of MS. Notes that the spirochetes may not necessarily be of the genus Borrelia burgdorferi. The chance that 100% of MS patients would also have Lyme neuroborreliosis is astronomically small – about one in 1000^10, a smaller chance to find a speck of dust lost in the Universe. Epidemiologically speaking, Brorson’s findings are near-absolute proof that MS is caused by spirochetal bacteria.

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2004 (Switzerland): Chronic Lyme borreliosis at the root of Multiple sclerosis – is a cure with antibiotics attainable? 

Synopsis: Notes that worldwide, MS prevalence parallels the distribution of the Lyme disease pathogen Borrelia burgdorferi, and in America and Europe, the birth excesses of those individuals who later in life develop MS, exactly mirror the seasonal distributions of Borrelia transmitting Ixodes ticks. No other disease exhibits equally marked epidemiological clusters by season and locality. Cites research whereby spirochetes were found in the brains of MS patients as early as 1928, and that in over 250 control cases of diversified diseases there never were spirochetes found. Notes that this research has been successfully replicated decades later by different scientists. Points out that a considerable body of clinical evidence supports the concept that cystic L-forms of Borrelia Burgdorferi may cause MS. Dismisses skepticism towards this concept with science-based arguments. Dismisses the hypothesis of genetic origin of MS using scientific research data. Includes graphs showing a direct correlation between the number of MS patients and the number of ticks transmitting Lyme disease. Dismisses the “environmental toxin” hypothesis of MS using scientific research data. Explains how Borrelia could cause all MS symptoms. Recommends trials with antibiotics for MS patients.

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2009 (Romania): Controversies in late Neuroborreliosis and Multiple sclerosis – case series

Synopsis: Found a significant percentage of people diagnosed with MS in fact having neuro-Lyme. Concludes that it is probable that MS is caused by an infectious agent and recommends testing MS patients for Lyme disease.

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Connection between the number of ticks transmitting Lyme disease and the birth excess of MS patients; Read Here:

http://www.owndoc.com/uploads/ms-borrelia-graph11.png http://www.owndoc.com/uploads/ms-borrelia-graph2.png

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The geographic mortality rate for MS is nearly identical to that of Lyme disease? With near-identical hot-spots for both diseases, even in areas with a low population density (but high tick density); Read Here:

http://owndoc.com/uploads/2010/09/ms-is-lyme.png http://www.esri.com/news/arcwatch/1007/spatial-patterns.html

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Even the Iranians have documented a link between Lyme-Borreliosis and Multiple Sclerosis. 24 of 61 MS patients tested positive with an IgM Western Blot for Borrelia burgdorferi. 

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Minocycline appears to be a novel antibiotic agent in the treating of Multiple Sclerosis; Read Here: 

http://owndoc.com/pdf/MS-lesions-minocycline.pdf

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Spirochetes can be cultured from people with suspected Lyme-Borreliosis. 

This individual is NOT a scientist (his research field is psychotherapy) but he has been greatly helped by advice from a scientist with considerable laboratory experience. The five individuals who had their blood cultured for the Lyme spirochete were all diagnosed with Chronic Fatigue Syndrome; two testing negative for Lyme on the ELISA test, one testing equivocal.

http://www.counsellingme.com/microscopy/bskculture.html

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There is also a lot of hard evidence that Fibromyalgia, Parkinson’s, CFS, Lupus, Crohn’s ME, Pick’s disease (FTD, Frontotemporal dementia), Alzheimers’ disease and ALS (Amyotrophic Lateral Sclerosis) are also caused by spirochetal bacteria. 

14 out of 16 deceased Alzheimer’s patients had living Treponema spirochetes in their brain. http://www.ncbi.nlm.nih.gov/pubmed/11929559?ordinalpos=10&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum

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Lou Gehrig had a house in Lyme, CT where Lyme Disease is most endemic.

http://www.als-cure.com/als-lyme/lou-gehrig-had-a-house-in-lyme/

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There are many case files and studies as of recent that provide evidence that the Lyme Spirochete can hide from the immune system and engender chronic morbidities; that mimic that of other neurological disease and in fact are the causative reason for the existence of these diseases.

Read Studies

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Lyme and CFS: https://ticktalkireland.wordpress.com/lyme-links/lyme-or-m-e/

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Lyme and ALS: https://ticktalkireland.wordpress.com/lyme-links/lyme-als/

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Lyme and Autism: https://ticktalkireland.wordpress.com/lyme-links/lyme-autism/

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Lyme and Alzheimers: https://ticktalkireland.wordpress.com/lyme-links/lyme-alzheimers/

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Lyme and Misdiagnosed MS: https://ticktalkireland.wordpress.com/lyme-links/lyme-ms/ http://www.lymeinfo.net/multiplesclerosis.html

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Criminal Charges; Falsifying diagnostics and Patents

Now I would like to talk about the criminal charges of the CDC, Yale and falsifying of the diagnostics. Yale has patents on diagnostic tests that are far more accurate than Lyme Deadborn test (15%) and even then they knew that Lyme disease should be perceived as a relapsing fever organism, undergoing antigenic variation. Hence, sufferers will only produce new igM bands if the Lyme spirochete is still alive and not killed by antibiotics or what ever else you use for eradicating bacteria.

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Allan Steere also wrote in that same 1986 report, basis of the 1990 CDC case definition

band 41 to diagnose Lyme;; just rule out syphilis: That is important to remember: You only need band 41, or the anti flagellar antibody and the triad of symptoms to diagnose Lyme with common sense rule. Molecular characterization of the humoral response to the 41-kilodalton flagellar antigen of Borrelia burgdorferi, the Lyme Disease spirochete.

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The earliest humoral response in patients infected with Borrelia burgdorferi, the agent of Lyme disease, is directed against the spirochete's 41-kDa flagellar antigen a SPECIFIC band in the Western Blot Lyme test. In order to map the epitopes recognized on this antigen, 11 overlapping fragments spanning the flagellin gene were cloned by polymerase chain reaction and inserted into an Escherichia coli expression vector which directed their expression as fusion proteins containing glutathione S-transferase at the N terminus and a flagellin fragment at the C terminus. Affinity-purified fusion proteins were assayed for reactivity on Western blots (immunoblots) with sera from patients with late-stage Lyme disease. The same immunodominant domain was bound by ser a from 17 of 18 patients. This domain (comprising amino acids 197 to 241) does not share significant homology with other bacterial flagellins and therefore may be useful in serological testing for Lyme disease.

http://www.ncbi.nlm.nih.gov/pubmed/1894359

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US patent 5, 618, 533 - Yale: detects, early, late, neurological, and every other possible kind of Lyme outcome and that it detects 94.4% of the cases , which means it is the closest possible method we could possibly have to detect Lyme ("should be 100% of the 57 cases," says the FDA, verbatim), and it should be noted that this was a specific method and does not test any other flagellins. When the FDA says "sensitivity," they really mean "LIMIT OF DETECTION" and refer to the METHOD and not the "CASES." “Accuracy” addresses cases.

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Yale and their patent would solve these problems, however they did not use this method to qualify LYMErix, their other patent, which is the essence of this False Claims Act. Moreover, the other Borrelia in North America and Europe, at least, such a method should be developed and the NIH agrees with this.

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The CDC needs to fund the right people willing to research Lyme and all of its forms. However, they still deny that cyst form of the Lyme spirochete exists because it would then be much easier for Lyme to evade immune response and their antibiotic guidelines. Lyme gets about $20 million/year in funding? That may sound like a lot but to put it in prospective: Cancer gets over $6 BILLION/year. Sleep Research gets $235 million/yr. AIDS gets $3.5 BILLION/yr. Smallpox $31million/yr. And that's just in taxpayers' NIH funds. Smallpox, which the CDC declares is eradicated (no cases since 1977) still gets $30 million/yr in funding. A disease that doesn't even exist anymore gets more research funds than Lyme. And LYME is more prevalent than AIDS and the fastest growing infectious disease in the world! The NIH website can verify each of these numbers. And that's just in taxpayer money. Privately, OHSU just received a $100million gift for cancer research. Bottom line, until there's REAL MONEY in Lyme, NOTHING will be done. Seems to me that the CDC and the IDSA are evading and whitewashing much of Alan MacDonald's research. Why the unwillingness to fund?

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MacDonalds; Breakthrough Lyme Research being whitewashed by the CDC; Read Here:

http://alzheimerborreliosis.net/research/

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http://alzheimerborreliosis.net/wp-content/uploads/2012/10/Teaching-file-Biofilms-of-Borrelia-burgdorferi.pdf

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https://www.lymedisease.org/the-rest-of-the-story-trials-and-tribulations-of-getting-borrelia-biofilms-acccepted-for-publication/

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https://www.youtube.com/watch?v=1ojq_2-HlNg

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Contd. Criminal Charges; Falsifying diagnostics, Patents and Lyme Vaccine

I'm am not anti-vaccine, I'm simply a skeptic who has researched the obfuscation of vaccines and their political agendas.

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The CDC and Allen Steere knew that Lyme was a relapsing fever organism. It has largely been known since the 80's. It means that the spirochete undergoes antigenic variation. They knew that sufferers would only produce new iGM bands if the spirochete was still alive in the host. They even said that all you need is band 41 to diagnose Lyme (Mentioned Above); just rule out syphilis. They knew this and then labeled Lyme as largely undetectable, hard to get and easy to treat!

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The CDC conducted fraud regarding the Dead-born diagnostics and are reluctant to fund projects that jeopardize their ridiculous guidelines.

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Initially: Lyme - Relapsing fever organism; causes various illnesses. Later on: Vaccine candidate; early set of trials; now a non-disease, easily treatable. They then took it off the market and the FDA sued for anti-trust. They simply redefined Lyme to pass off their vaccine.

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All in all, the Infectious Disease Society of America and the CDC have committed research fraud to falsify the current, Dearborn case definition. And in order to falsify the outcomes of the OspA Vaccines; Lymerix Vaccine, which was pulled not because of the simply trivial statement of anti-vaccine but because of the FDA ultimatum to the manufacturer. Steere and the CDC wanted substantial financial rewards from a monopoly on all tick borne diseases, vaccines and test kits. Their falsification of the case definition and treatment guidelines have left 85% of actual Lyme sufferers unable to obtain diagnosis, treatment, or insurance coverage for their illness. They had significant conflict of interests and looked to collude with various organizations namely the ALDF where they would use their fraudulent recombinant kits.

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THEY CLAIMED Lyme was not a relapsing fever organism, when they KNEW it was and whistle-blowers and documents in the public domain have showed that these people knew it.

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So, the made Lyme largely undetectable despite the obvious test patents that are very accurate and sensitive to various strains. No matter how crazy this sounds these people sought to profit off of these vaccines. People have gotten terribly sick off of the Lymerix vaccine and it is well documented. If you have a test that is only 15% accurate (detects 15% of cases), the defendant would have an 85% effective vaccine!! They also used propagandizing tactics and discredited anyone who became sick and suffered from Lyme symptoms after getting the vaccine.

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The vaccine was an immunesupressive T-LR21 agonist; Please read this study: http://www.ncbi.nlm.nih.gov/pubmed/10865170

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Diagnostics                             

Think of all bands on the Western Blot Lyme test as Antibodies, some are highly SPECIFIC for Lyme, others cross react with various infections, while still others are not that important. A patient can be positive or quadruple positive for an antibody on the Western Blot test, namely band, 18 kDa, 21 kDa, 23 kDa, 28 kDa, 30 kDa, 31 kDa, 34 kDa, 39 kDa, 41 kDa, 45 kDa, 58 kDa, 66 kDa, 83-93 kDa. Bands in Bold are HIGHLY SPECIFIC Lyme Antibodies. THe Western Blot features an IgM test (Acute infection) and IgG test (Chronic or Long-term Infection).

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The IgM response, +, ++, +++, or ++++ for Band 41 on the Western Blot test before the 1994 case definition is all you needed to diagnose Lyme and get the appropriate treatment because it was easy to rule out all the other infections that cross reacted with this specific band.

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During arthritis from Lyme a new IgM response most likely develops into band 31, which I was positive for. But the new case definition based on research fraud and falsification of diagnostics that is still CURRENTLY BEING USED recommends that an IgG immunoblot be considered positive if five of the following 10 bands are present: 18 kDa, 21 kDa (OspC)*, 28 kDa, 30 kDa, 39 kDa, 41 kDa, 45 kDa, 58 kDa, 66 kDa and 93 kDa.

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I tested positive for IgM 18 kDa (Specific), 31 kDa (specific), 39 kDa IND (specific); IND means highly likely a single positive, 41 kDa ++++, 83-93 (Specific) IND. I was NEGATIVE by CDC/NYS results, Positive by IGENEX IgM Rsults.

I tested positive for IgG 31, 41 kDa ++, 58 kDa +. I was CDC NEGATIVE, IGENEX RESULT IND.

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I had highly specific Lyme bands, symptoms that classically represented Lyme, a tick that I knew I was bitten by and yet even with a positive result for IGENEX I was refused coverage and treatment. Remember prior to 1994, all that was needed was 41 kDa band to diagnose.

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Conclusion

In conclusion, there is growing scientific evidence that chronic Lyme disease does exist, and that this clinical condition is related to persistent infection with B. burgdorferi as shown by microbiological and molecular studies. Persistent infection occurs in animal models and humans because the Lyme spirochete is able to evade both the host immune response and short-course antibiotic therapy to establish chronic infection in protected tissue sites, much like TB. This chronic infection leads to persistent musculoskeletal, neurologic and cardiac symptoms that are the hallmark of chronic Lyme disease. By contrast, the leading theory for persistent symptoms owing to 'post-Lyme syndrome', namely an autoimmune response triggered by the eradicated spirochetal infection, has not been supported by scientific evidence.

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Quote from Alan MacDonald:

We live in an arena where there are political, as well as scientific and medical discussions. And I think that scientific and medical discussions are important, and political discussions sometimes prevent us from getting very far with our science and our medicine. As long as people enter an opportunity for discussion and dialog with preconceived notions about what they will and will not embrace as an idea, you have a political weight that is dragging down the level of the opportunity to learn from discussion or from listening to patients.

If people decide that there is no such thing as maternal fetal transmission in Lyme disease, then they can't learn very much when they read my paper, because my paper does show that there is tissue evidence of the infection in newborns that were stillborn, or fetuses that didn't make it to full term.

And there are opportunities to read other papers and not understand how dormant conditions can exist for twenty or thirty years, such as the ACA model in Europe. If you enter the discussion with a preconceived notion of what you will or will not take away from the discussion, you can't learn. So politics is an unfortunate mindset, where the mind is closed before the discussion begins. And that's where a lot of, I think, the politics of discord come from in discussing this, or any other infection.

You know, in the early days of HIV infection, there was a certain reluctance to see it as a public health problem that we know now HIV infections are, in a worldwide basis. But back in the early eighties, when I was beginning my practice, there wasn't a lot of open mindedness about the need to treat HIV as a medical problem. It was a political problem for people who came from Haiti, or for people who lived in San Francisco who happened to be gay. It was a political problem.

Now, we have gotten past that and we see that HIV is a chronic infection. It's a medical issue, not a political issue anymore. Sometimes it takes a long time for politics to exit, and for medicine and science to get back on an even keel."

Some more studies about Chronic Lyme

https://durayresearch.wordpress.com/our-work/other-topics/

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http://www.ncbi.nlm.nih.gov/pubmed?term=MacDonald%20AB[Author]&cauthor=true&cauthor_uid=16675154

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http://www.ncbi.nlm.nih.gov/pubmed/23346260